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The mutation of the MLPH gene leads to a dilution of the intensity of the coat colour. The black coat becomes blue, the orange becomes cream and the chocolate coat is diluted to lilac.

Feline hypertrophic cardiomyopathy (HCM) is characterised by left ventricular hypertrophy. Symptoms are variable from individual to individual and include heart failure, fatigability and in some cases death of the animal. The causative mutation is different between breeds. HCM1 is specific for the Maine Coon.

Feline hypertrophic cardiomyopathy (HCM) is characterised by left ventricular hypertrophy. Symptoms are variable from individual to individual and include heart failure, fatigability and in some cases death of the animal. The causative mutation is different between breeds. HCM4 is specific for the Sphynx.

Feline spinal muscular atrophy is a disease of the nervous system. It is characterised by gait abnormalities due to the loss of motor neurons in the lower spinal cord and atrophy of the hind limb muscles. Affected kittens show the first signs of SMA at around 3-4 months of age.

The ASIP protein interacts with the melanocortin-1 receptor to alternate between black and red pigments, creating a banding pattern in the hair. Mutations in the ASIP gene prevent this change from occurring, resulting in uniformly coloured hair. The colour of the cat's coat also depends on complex interactions with other genes.

Mucopolysaccharidosis VI is a lysosomal storage disease, the symptoms of which can appear as early as 6 weeks of age. These include facial dysmorphia, reduced flexibility, growth retardation, loss of mobility of posterior limbs, joint problems and corneal opacification.

Gangliosidosis is characterised by muscle tremors and loss of motor control. The first symptoms are observed at around 6-8 weeks of age and start with slight tremors that result in feeding difficulties and lack of coordination.

This test is also recommended for Bombay and Australian Mist as part of the breeding program established by the Flemish government (see official program).

Feline hypertrophic cardiomyopathy (HCM) is characterised by left ventricular hypertrophy. Symptoms are variable from individual to individual and include heart failure, fatigability and in some cases death of the animal. The causative mutation is different between breeds. HCM3 is specific for the Ragdoll.

This test is also recommended for the Ragamuffin as part of the Flemish government breeding program (see the official program).

The Devon Rex's distinctive curly coat is the result of a genetic mutation in the KRT71 gene, which is essential for hair formation. This mutation causes an alteration in the structure of the hair, giving the Devon Rex its characteristic appearance, with tight curls and a unique texture. All Devon Rexes analysed have two copies of this mutation, which means that it is fixed in the breed and determines the curly phenotype. This mutation is specific to the Devon Rex and has not been observed in other rexoid breeds or hairless varieties, highlighting its uniqueness within the breed.

GM1 gangliosidosis is a fatal genetic disease caused by a deficiency of the enzyme β-galactosidase. This enzyme normally helps break down certain complex lipids called gangliosides, particularly GM1 ganglioside. In the absence of this enzyme, these compounds progressively accumulate within cells, especially in nervous tissue. The disease generally manifests around the age of 3 months and reaches its terminal stage at around 9 to 10 months, with the onset of blindness and epileptiform seizures. Affected animals exhibit an unsteady gait, loss of coordination, tremors, and progressive muscle weakness, leading to severe neurological decline.