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Hypokalaemia is a condition related to low serum potassium and high creatine phosphokinase. Symptoms include episodes of muscle weakness usually in the neck but sometimes only in the limbs. As a result, affected cats may have problems walking and holding their heads properly.

Cystinuria is caused by a lack of renal reabsorption of the amino acid cystine. This excess of urinary cystine causes crystals to form, which can lead to the formation of stones in the kidney and/or bladder. Cats with cystinuria suffer from repeated inflammation of the urinary tract.

In domestic cats, the most common blood groups are A and B, while a third rare blood group, AB, is also recognized. This terminology can be confusing because type AB is not the result of a combination of groups A and B.

In Ragdoll cats, three genetic mutations allow determination of the blood group as well as the cat’s status regarding the b allele. This test is particularly important due to the risk of neonatal isoerythrolysis: the colostrum of a mother with blood group B can be fatal to a kitten with blood group A or AB.

This test analyzes mutations specific to the Ragdoll breed and is not applicable to other breeds.

Congenital myasthenic syndrome, also known as spasticity, is a disease causing congenital muscle weakness. Affected cats frequently adopt a characteristic squirrel-like position by resting their front paws on an object. Signs of the disease appear at about 3 weeks of age. The disease can progress slowly but affected individuals usually die before 2 years of age.

GM2 gangliosidosis is an inherited disease caused by a deficiency of the enzyme β-hexosaminidase, leading to the accumulation of GM2 gangliosides in the nervous system.
In Korat cats, the first signs appear as early as 4 to 6 weeks of age with fine head tremors.
The disease progresses quickly with coordination problems, falls, and abnormal gait.
Affected kittens show severe deterioration over the following months.
Sadly, the outcome is fatal before 8 months of age.

 

Progressive retinal atrophy (PRA) linked to the AIPL1 gene is a hereditary genetic disease described in Persian cats. It causes rapid degeneration of retinal photoreceptors, with the onset of cell loss as early as 5 weeks of age and severe involvement by 16 weeks. The first clinical signs include an early decrease in night vision, followed by loss of daytime vision.

This test is also recommended for the Exotic Shorthair as part of the Flemish government breeding program (see the official program).

Sex-linked orange coat colour in cats is associated with an approximately 5 kb deletion in the ARHGAP36 gene, located on the X chromosome. This variant causes abnormal ARHGAP36 expression in melanocytes, reducing the production of eumelanin, the black-brown pigment, and promoting pheomelanin, which is responsible for orange coloration. Males, which have only one X chromosome, are orange when they carry the O variant, whereas they do not show this colour when the variant is absent. In females, two copies of the variant produce an orange coat, while a single copy leads to mosaic expression of orange and non-orange areas, resulting in tortoiseshell or calico coat patterns.

Multidrug Resistance 1 is a genetic mutation in a protein that transports drugs out of the brain. This mutation causes sensitivity to certain drugs, including ivermectin and loperamide. Because of this lack of drug clearance from the brain, affected cats experience neurological symptoms such as ataxia or even death. The MDR1 mutation has currently been identified in the breeds listed below; however, the test can be conducted in any breed.

This test is also recommended for the Javanese (see the official program) and Madarin (see the official program) as part of the Flemish government breeding program.

Factor XI deficiency is a genetic disorder affecting the coagulation process. Symptoms of this condition are excessive bleeding following surgery or trauma.

A mutation in the KRT71 gene is responsible for the virtual absence of hair in the Sphynx. This genetic alteration disrupts normal hair production, leading to almost total loss of coat.