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This panel includes the essential tests to be performed for the American Bulldog: ichthyosis (DOG-ARCI), hyperuricosuria (DOG-HUU) and Neuronal ceroid lipofuscinosis type 10 (DOG-NCL10)

This profile includes the specific tests to be performed for Belgian Shepherds: spongy degeneration with cerebellar ataxia type 1 (DOG-SDCA1), spongy degeneration with cerebellar ataxia type 2 (DOG-SDCA2), X-linked hypohidrotic ectodermal dysplasia (DOG-XHED) and cardiomyopathy and juvenile mortality (DOG-CMJM-BB).

This panel includes the specific tests to be performed for the American Bulldog: ichthyosis (DOG-ARCI), canine multifocal retinopathy 1 (DOG-CMR1), hyperuricosuria (DOG-HUU) and Neuronal ceroid lipofuscinosis type 10 (DOG-NCL10)

Cerebellar degeneration—myositis complex is a degenerative encephalopathy. Clinical signs in affected dogs are highly variable and may include muscle weakness, generalized ataxia and hypermetria. The age of onset of neurological signs is between 10 weeks and 6 months.

Dogs with photoreceptor dysplasia appear normal up to 10 months old but develop vision problems by 3 years, such as slow pupillary reflexes and difficulty seeing in familiar places. Fundus abnormalities include vessel loss and retinal thinning, leading to eventual blindness before age five.

Palmoplantar Keratoderma is a genetic condition that typically appears between 10 weeks and 1 year of age. Affected dogs develop painful thickening of the paw pads, with keratinous proliferations and fissures localized to the areas in contact with the ground. These fissures can lead to secondary infections and may cause lameness or reluctance to walk. The nails and other areas of the skin do not appear to be affected, and no other associated skin signs have been reported.

This cardiomyopathy leads to death without apparent cause at around 6-8 weeks of age in the Belgian Shepherd. Symptoms of this condition vary from individual to individual and include: vomiting, dyspnoea, enlarged heart, difficulty in locomotion, tremors.

GM1 gangliosidosis is a fatal genetic disease caused by a deficiency of the enzyme β-galactosidase. This enzyme normally helps break down certain complex lipids called gangliosides, particularly GM1 ganglioside. In the absence of this enzyme, these compounds progressively accumulate within cells, especially in nervous tissue. The disease generally manifests around the age of 3 months and reaches its terminal stage at around 9 to 10 months, with the onset of blindness and epileptiform seizures. Affected animals exhibit an unsteady gait, loss of coordination, tremors, and progressive muscle weakness, leading to severe neurological decline.

Degenerative myelopathy is a neurodegenerative disease that progresses slowly and has a late onset (8 years or more). The initial clinical sign is characterised by hind limb ataxia. As the disease progresses, the frequently observed asymmetric weakness rises to affect the thoracic limbs, leading to paraplegia.

Exercise induced collapse is a neuromuscular disorder characterised by muscle weakness, incoordination and life-threatening collapse after intense exercise. Five to fifteen minutes of intense exercise causes dogs with this syndrome to develop a staggering gait and loss of control of the hind limbs. The episode may extend to all four limbs. Collapsing episodes usually last 5-10 minutes, and after 30 minutes there is often a full recovery, but episodes are sometimes fatal. This test is patented and subcontracted to the patent holding laboratory.