Search results for: 'WA 0812 2782 5310 Biaya Renovasi Rumah Minimalis 9 X 15 Tegalrejo Yogyakarta'

This profile includes the specific tests to be performed for Belgian Shepherds: spongy degeneration with cerebellar ataxia type 1 (DOG-SDCA1), spongy degeneration with cerebellar ataxia type 2 (DOG-SDCA2), X-linked hypohidrotic ectodermal dysplasia (DOG-XHED) and cardiomyopathy and juvenile mortality (DOG-CMJM-BB).

This panel includes the specific tests to be performed for the German Shepherd Dog: achromatopsia (DOG-ACHMB), Scott syndrome (DOG-CSS), coat length (DOG-LH), multidrug resistance (DOG-MDR1), mucopolysaccharidosis VII (DOG-MPS7), dwarfism (DOG-NAN), renal cystadenocarcinoma and nodular dermatofibrosis (DOG-RCND) and X-linked hypohidrotic ectodermal dysplasia (DOG-XHED).

GM1 gangliosidosis is a fatal genetic disease caused by a deficiency of the enzyme β-galactosidase. This enzyme normally helps break down certain complex lipids called gangliosides, particularly GM1 ganglioside. In the absence of this enzyme, these compounds progressively accumulate within cells, especially in nervous tissue. The disease generally manifests around the age of 3 months and reaches its terminal stage at around 9 to 10 months, with the onset of blindness and epileptiform seizures. Affected animals exhibit an unsteady gait, loss of coordination, tremors, and progressive muscle weakness, leading to severe neurological decline.

Congenital myasthenic syndrome is an inherited neuromuscular disease observed in the Jack Russell Terrier. Affected puppies appear normal until 6 weeks of age, then gradually develop generalized muscle weakness, walking briefly for 10 to 15 steps before stopping or lying down. Treatment with anticholinesterase drugs leads to temporary improvement, but resistance to the treatment often requires euthanasia.

In the Jack Russell Terrier, congenital myasthenia is most commonly caused by the 729dup mutation. However, under the breeding regulation of the Flemish government, it is the 1508dup mutation that must be tested for in both the Jack Russell Terrier and the American Staffordshire Terrier. This test specifically detects the 1508dup mutation.

This profile includes the essential tests to be performed for Rottweiler: juvenile laryngeal paralysis and polyneuropathy (DOG-JLPPTNR), leukoencephalomyelopathy (DOG-LEMPR), and neuro-axonal dystrophy (DOG-NAD).

Leukoencephalomyelopathy is a neurodegenerative disease characterised by a progressive loss of mobility and coordination of the limbs. The first symptoms usually appear between 1 and 4 years of age.

Neuroaxonal dystrophy is a genetic neurodegenerative disease. Symptoms include various neurological symptoms such as abnormal gait, loss of balance, head tremors and involuntary eye movements (nystagmus).

This pack includes the recommended tests defined by the Flemish breeding program for Rottweiler: Degenerative myelopathy 2 (DOG-DM2) and Von Willebrand disease type 1 (DOG-VWD1). The Flemish breeding program also makes the tests included in our package mandatory DOG-ROTT-VL.

This package includes the mandatory tests defined by the Flemish breeding program for Rottweiler: Juvenile Laryngeal Paralysis and Polyneuropathy (DOG-JLPPTNR), Leukoencephalomyelopathy (DOG-LEMPR), Neuroaxonal dystrophy (DOG-NAD) and LOXHD1-related deafness (DOG-EOAD-ROTT). The Flemish breeding program also recommends the tests included in our package DOG-ROTT-VL+.

Congenital deafness in Rottweilers is a genetic disorder causing bilateral hearing loss from the first weeks of life. Affected puppies show profound deafness, without other apparent clinical signs.

This condition is associated with a mutation in the LOXHD1 gene, which plays a key role in the function of the cochlear hair cells.