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Sex-linked orange coat colour in cats is associated with an approximately 5 kb deletion in the ARHGAP36 gene, located on the X chromosome. This variant causes abnormal ARHGAP36 expression in melanocytes, reducing the production of eumelanin, the black-brown pigment, and promoting pheomelanin, which is responsible for orange coloration. Males, which have only one X chromosome, are orange when they carry the O variant, whereas they do not show this colour when the variant is absent. In females, two copies of the variant produce an orange coat, while a single copy leads to mosaic expression of orange and non-orange areas, resulting in tortoiseshell or calico coat patterns.

The mutation of the MLPH gene leads to a dilution of the intensity of the coat colour. The black coat becomes blue, the orange becomes cream and the chocolate coat is diluted to lilac.

In donkeys, dominant white corresponds to complete depigmentation: the animal is born with a white coat, pink skin and dark eyes. A variant in the KIT gene, c.662A>C, was identified in a donkey showing this phenotype and is considered a candidate variant associated with dominant white. The genetic test can be used to detect this variant and identify animals that carry it. However, the available data remain preliminary, as this variant was observed in only one white donkey in the study.

Hypocatalasia (or acatalasemia) is a rare genetic disorder caused by a mutation in the CAT gene, leading to a severe deficiency of catalase. This enzyme normally protects tissues against oxidative stress. The c.979G>A (p.Ala327Thr) mutation was first identified in a Beagle colony and subsequently detected in pet Beagles. A homozygous dog notably developed oral cavity gangrene with tooth loss at a young age. The disease may remain clinically silent for a long time, which highlights the importance of genetic screening in breeding dogs.
This test is also recommended for the Basset Hound as part of the Flemish government breeding program (see the official program).

The PPARD gene is involved in mechanisms related to energy metabolism, fat utilisation and muscular endurance. In racing pigeons, a recent study suggests that certain variations in this gene may be linked to race performance, particularly return speed and ranking coefficient. The CC genotype for the PPARD c.*271C>T marker is the one that stands out most favourably in the study, notably because it was observed in pigeons taking part in a larger number of races. This test can therefore provide complementary information to guide the selection of breeding birds, without replacing the assessment of the pigeon, its lineage, training and actual racing results.